Depression
1. Factors that are likely to play a role include:a) genetics; changes in the brain’s neurotransmitter levels; environmental factors; psychological and social factors; additional conditions, such as bipolar disorder 2. Depression is almost twice as likely to affect women than men and tends to have different contributing causes in women than it does in men.
a) Contributing factors in women include reproductive hormones, a differing female response to stress, and social pressures that are unique to a woman’s life experiences.
There are a multitude of genetic, hormonal, psychological, and social factors that come into play when citing the cause of depression in women. b) Biology and Hormones
Biologically speaking, depression runs in families – with scientific evidence that some genetic makeups are more prone to depression, whereas some genetic makeups are more resistant to it.
Issues with pregnancy, fertility, perimenopause, menopause, and menstrual cycles increase women’s risk factors of developing depression. Most of these are due to hormonal imbalances and rapid fluctuations in reproductive hormones. c) Psychological Causes
Women are more prone to psychological causes of depression than men. With a tendency to be more emotional, women are more likely to rehash negative thoughts during bouts of depression. While it is a normal response to cry, talk with friends, and rehash why it is you are in your depressive state, research has shown that ruminating about depression can cause it to last longer and even make it worse. In contrast, men tend to distract themselves from their depressive state – which has been shown to reduce the duration of symptoms. Additional psychological factors that tend to affect women over men are negative body images and stress-induced depression. Women are more prone to stress than men because their increased levels of progesterone have been shown to prevent stress hormones from leveling out. Negative body image issues usually begin in adolescence and seem to be correlated with the onset of puberty in women. d) Social Causes
Coping skills, choice of relationships, and lifestyle choices affect women differently than men. As a woman, you are more likely to develop depression from marital or relationship problems, work-life balance issues, financial troubles, and stressful life events, including the loss of a loved one.
Depression is diagnosed by first ruling potential underlying medical conditions and medications that might be to blame.
Common side effects of antidepressant medication include Nausea, Headaches, Sleep disturbances [These make depression worse and contribute to Dementia Syndrome symptoms] e) Depression in women can have misdiagnosis rates as high as 50%. f) In addition to medications and therapy:
Don’t keep your feelings bottled up –[yet the society is filled with many messages to tell you only put on a happy face, smile, look on the bright side, don't be negative, etc.]
Exercise regularly
Get enough sleep [medication works against this] 3. Major depression is the leading cause of disability worldwide. 4. A lot of the logic above has holes in it and doesn't hold water. It shows that everything connected to the medical industry is a big guessing game with misplaced aim, blame and gain.
Hallucinations
1. Similar to my take on who's crazy and who's not, here's my balanced take on hallucinations.If someone is considered sick or otherwise mentally ill just because they are seeing things that are not there (visual hallucinations), then so also should someone be considered sick or mentally ill for not seeing things that are there (dissociative rose-colored mental blindness). In other words, if someone is considered sick for seeing things we can't see, then aren't we also sick for not being able to see something that someone else can see? No, I'm not talking about hallucinations, I'm talking about not being able to see the truth, the lie, the okie doke and many other things obvious to someone else, such as the causes and effects of Maafa Racism's nonstop onslaught. Bottom Line: In societies, when you are in the minority, you are considered abnormally sick or abnormally genius.
In civilizations, both the minority and the majority are continuously assessed to understand if it's one or the other or both.
The ultimate solution (pure understanding) is often a set of variables and/or a set of sets of variables). 2. Visual hallucinations have numerous etiologies.
Numerous hypotheses have been suggested to explain the cause of visual hallucinations. : psychophysiologic (i.e., as a disturbance of brain structure), psychobiochemical (as a disturbance of neurotransmitters), and psychodynamic (as an emergence of the unconscious into consciousness). Visual hallucinations can be the result of all 3 processes, given the interplay among disturbances of brain anatomy, brain chemistry, prior experiences, and psychodynamic meaning. To date, no single neural mechanism has explained all types of visual hallucinations; however, the similarity of visual hallucinations seemingly associated with diverse conditions suggests a final common pathway.. a) Irritation (e.g., seizure activity) of the primary visual cortex causes simple elementary visual hallucinations, while irritation of the visual association cortices causes more complex visual hallucinations.
b) Lesions that cause deafferentation of the visual system may lead to cortical release phenomenon, including visual hallucinations.
c) A multitude of lesions can cause this loss of input and inhibit other cognitive functions. Of note, visual hallucinations may be induced by prolonged visual deprivation. There is strong support to the idea that the simple loss of normal visual input is sufficient to cause visual hallucinations.
d) Finally, due to its role in the maintenance of arousal, the reticular activating system has been implicated in the genesis of visual hallucinations. Lesions of the brainstem have led to visual hallucinations (as in peduncular hallucinosis). Further, visual hallucinations are common in those with certain sleep disorders, and occur more frequently in those who are drowsy. The observation that visual hallucinations occur more frequently in those who are drowsy (even in the absence of frank sleep pathology) suggests that the reticular activating system plays a role in visual hallucinations, although the precise mechanism has not yet been established. 3. Conditions That Can Cause Visual Hallucinations
a) Psychosis (schizophrenia/schizoaffective disorder) - While the majority of hallucinations reported in primary psychotic disorders are auditory, they may also be visual, olfactory, tactile, or gustatory.
Visual hallucinations in those with schizophrenia tend to involve vivid scenes with family members, religious figures, and animals. The hallucinations are usually described as colorful and involving normal-sized people and objects. 4. Delirium is a syndrome that involves an acute disturbance of consciousness as well as a diminished ability to sustain attention, is caused by myriad medical conditions, metabolic disturbances, infections, drug effects, and intracranial processes. It is often manifest by symptoms (e.g., hallucinations and delusions) that are suggestive of a primary psychotic illness. Among those with delirium, visual hallucinations are the most common type of hallucination. 5. Dementia with Lewy bodies (DLB) is the second most common form of dementia after dementia of the Alzheimer's type. Prominent symptoms include parkinsonism, visual hallucinations, and cognitive fluctuations. Visual hallucinations occur in more than 20% of patients diagnosed with DLB.[This is low, thus not an indicative symptom]
The visual hallucinations in DLB involve seeing objects move when they are actually still and seeing complex scenarios of people and items that are not present.
b) Visual hallucinations also occur in up to half of patients with Parkinson's disease. These hallucinations are similar to those in patients with DLB, and can range from seeing a person or animal to seeing more complex, formed, and mobile people, animals, or objects. DLB and dementia associated with Parkinson's disease are “more similar than different” and noted that the major difference is in the timing of symptom onset (with motor symptoms preceding cognitive decline in Parkinson's disease and vice-versa in DLB).
c) Posterior cortical atrophy is another neurodegenerative syndrome associated with visual hallucinations and parkinsonian symptoms. The chief pathologic features of posterior cortical atrophy are neurofibrillary tangles and senile plaques similar to those seen in dementia of the Alzheimer's type but with a distribution limited to the occipital and parietal lobes. Patients with posterior cortical atrophy may present with visual agnosia, anomia, apraxia, and features of Gerstmann syndrome. Neuroimaging typically reveals atrophy of the bilateral occipital, parietal, and posterior temporal lobes. 6. Charles Bonnet syndrome (CBS) involves the occurrence of visual hallucinations in the visually impaired. Any cause of visual impairment (including macular degeneration, glaucoma, cataracts, cerebrovascular disease, and tumors) may be associated with CBS.
b) The visual hallucinations in CBS have been described as clear and detailed; they often involve people, faces, animals, and inanimate objects. While patients may initially be unaware that these images are indeed hallucinations, one of the hallmarks of CBS is the eventual development of insight regarding their visual hallucinations. 7. Anton's syndrome is a rare condition in which patients with cortical blindness deny that they have visual loss. 8. Seizure-induced visual hallucinations have often been described as simple, brief, and consistent for each patient; they usually consist of small, brightly colored spots or shapes that flash. Content of the hallucination may be distorted in size or it may suddenly change shape, moving from a lateral field toward the center of the field of vision. Those that remain isolated in 1 visual field suggest seizure activity that originates on the contralateral side.
b) Since occipital seizures are frequently accompanied by a postictal headache, they may be difficult to distinguish from migraines.
c) While neurologic literature indicates that visual hallucinations associated with seizures are simple, there is a growing body of work that describes complex, formed visual hallucinations as ictal, peri-ictal, and intra-ictal phenomena. These symptoms may occur as part of a broader ictal psychosis that may feature delusions and paranoia, and may be indistinguishable from a primary psychotic disorder, especially if the seizures are of the complex partial type. The occurrence of complex visual hallucinations as a symptom of epilepsy can be a point of disagreement between neurologists and psychiatrists. 9. Migraine-induced visual hallucinations can be a classic aura of migraine as well as a less common manifestation (such as migraine coma and familial hemiplegic migraine). 10. Peduncular hallucinosis is characterized by visual hallucinations that arise following an infarct of the midbrain. The mechanism has been difficult to pinpoint since the literature features a wide variety of lesions. Lesions may involve the cerebral peduncles, but most often involve the reticular formation or its targets. 11. Sleep disturbance-induced hypnagogic hallucinations are visual and auditory perceptions that occur during sleep onset, while hypnopompic hallucinations occur on awakening. 12. Drug effects
a) Many drugs are labeled as hallucinogens because they alter perceptions, although true hallucinations are perceptions in the absence of any actual stimulus. 13. Tumors that lie along, or compress, the optic path may cause visual hallucinations. 14. Inborn errors of metabolism.
a) A handful of inborn errors of metabolism may cause visual hallucination. 15. Creutzfeldt-Jakob disease (CJD) is a fatal progressive neurodegenerative illness caused by central nervous system prion infection. Prominent symptoms typically include fatigue, anxiety, and personality change, with progression to dementia, ataxia, and myoclonus in the later stages. Symptoms of CJD may also include visual hallucinations. 16. How Can the Etiology of Visual Hallucinations Be Determined?
a) Given the broad variety of potential etiologies of visual hallucinations outlined previously, it is clear that an accurate diagnosis is required before effective treatment can be initiated. A thorough history and clinical examination are the most vital elements of a workup for visual hallucinations. An EEG is potentially the most revealing diagnostic study, since it can not only highlight seizure activity, but also detect delirium and CJD. An MRI might also help.
Protein Plaques
1. Protein plaques are a buildup of protein in certain areas of the body, including the brain. This is fundamentally caused by cellular malfunction which then results in disorders such as cancer, metabolic and neurodegenerative diseases and more. As always multiple processes and cell structures are involved. This includes endosomes, lysosomes, golgi appratuses and a host of other mechanisms in human cells to recycle amino acids and proteins. 2. Some researches think a cellular machine, called FERARI (Factors for Endosome Recycling And Retromer Interactions) might be responsible. 3. Some researches think a cellular component called the proteasome is responsible.. 4. “Proteasome inhibitor medication already exists and is currently being used to treat some cancers.” 5. Medication (toxins) are always implicated as causing disorders because that's what side effect means. Depression, hallucinations and protein plaques are common to many syndromes and diseases, yet these things are used to try to pinpoint a disease and prescribe a medication. This is the end game – to drain the person while draining their pockets and insurance money. The drug industry is the tail that wags the dog of the medical industry whose corrupt heart and soul are universities and their government parents. These messages are not about trying to understand these topics so as to become an expert of suboptimal logic, but to allow our spirit expert to show us that the logic of the experts we are following is deeply flawed and so are we for following. Humans in societies have a fundamental disconnect between their cellular homeostasis and the ecosystem, both being the same thing in different forms. We are earthlings of a Human Earth. If not, then stop saying Mother Earth. Stop saying man was formed of Earth. And if Eve is the mother of all living, then that makes her Mother Earth. And since humans were made in God's image, that makes God the same as Adam, Eve and Earth. There is only one way out of our death spiral and it does not involve relying on medication or other lifestyle methods put forth by madmen and madwomen who will do anything to avoid starvation, yet starvation is who they are starved of the essential goodness, thus starvation is their M.O. Magnum opus modus operandi.Sankofa to Sankhemet and Horus-Chem, Lord of the land of San and the new Sandemic authored by the New Age iterations Aquaria and Kepheus and the others at six more corners..
